Record Display for the EPA National Library Catalog


Main Title Effects of low levels of ozone and temperature stress /
Author Horvath, Steven M., ; Folinsbee., Lawrence J.
Other Authors
Author Title of a Work
Folinsbee, Lawrence J.,
CORP Author California Univ., Santa Barbara. Inst. of Environmental Stress.;Health Effects Research Lab., Research Triangle Park, N.C.
Publisher U.S. Environmental Protection Agency, Office of Research and Development, Health Effects Research Laboratory, Available through the National Technical Information Service
Year Published 1976
Report Number EPA-600/1-76-001; EPA-68-02-1723; PB252309
Stock Number PB-252 309
OCLC Number 02214455
Subjects Ozone--Physiological effect ; Temperature--Physiological effect ; Cardiovascular system ; Respiratory organs ; Heat--Physiological effect ; Respiration--Measurement
Additional Subjects Heat stress ; Ozone ; Cardiovascular system ; Respiratory system ; Toxicology ; Body temperature ; Heart rate ; Oxidizers ; Air pollution ; Toxicity ; Stress(Physiology) ; Toxic tolerances ; Humans ; Standards ; Environmental health ; Air pollution effects(Humans) ; Maximum permissible exposure
Internet Access
Description Access URL
Library Call Number Additional Info Location Last
EJBD ARCHIVE EPA 600-1-76-001 Headquarters Library/Washington,DC 05/13/2013
EJBD  EPA 600-1-76-001 Headquarters Library/Washington,DC 01/17/2014
EKBD  EPA-600/1-76-001 Research Triangle Park Library/RTP, NC 06/06/2003
ELBD  EPA 600-1-76-001 AWBERC Library/Cincinnati,OH 11/15/2016
NTIS  PB-252 309 Some EPA libraries have a fiche copy filed under the call number shown. 07/26/2022
Collation x, 84 pages : graphs ; 28 cm.
Cardiopulmonary and metabolic responses of 20 adult males (age 19-29) before, during and after a 2-hour exposure to either filtered air or 0.50 ppm ozone under four ambient conditions (25C, 45% rh; 31C, 85% rh; 35C, 40% rh; 40C, 50% rh) were determined. Exercise at 40% of the individual's V02 max was performed from 60-90 min of exposure. There were no cardiovascular changes due to ozone exposure but heart rate increased and stroke volume decreased with increasing heat stress. Rectal, mean body, and mean skin temperature also increased. There was a decrease in vital capacity and total lung capacity due primarily to a reduction of inspiratory capacity following ozone exposure. The combination of heat stress and ozone exposure resulted in significantly greater impairment of pulmonary function. The trachial-bronchial irritation caused by ozone reduces the vital capacity and maximum expiratory flow and this effect is more pronounced when the ozone exposure occurs in a hot environment.
Contract no. EPA 68-02-1723. Includes bibliographical references (pages 47-53).