Main Title |
Effects of low levels of ozone and temperature stress / |
Author |
Horvath, Steven M., ;
Folinsbee., Lawrence J.
|
Other Authors |
|
CORP Author |
California Univ., Santa Barbara. Inst. of Environmental Stress.;Health Effects Research Lab., Research Triangle Park, N.C. |
Publisher |
U.S. Environmental Protection Agency, Office of Research and Development, Health Effects Research Laboratory, Available through the National Technical Information Service |
Year Published |
1976 |
Report Number |
EPA-600/1-76-001; EPA-68-02-1723; PB252309 |
Stock Number |
PB-252 309 |
OCLC Number |
02214455 |
Subjects |
Ozone--Physiological effect ;
Temperature--Physiological effect ;
Cardiovascular system ;
Respiratory organs ;
Heat--Physiological effect ;
Respiration--Measurement
|
Additional Subjects |
Heat stress ;
Ozone ;
Cardiovascular system ;
Respiratory system ;
Toxicology ;
Body temperature ;
Heart rate ;
Oxidizers ;
Air pollution ;
Toxicity ;
Stress(Physiology) ;
Toxic tolerances ;
Humans ;
Standards ;
Environmental health ;
Air pollution effects(Humans) ;
Maximum permissible exposure
|
Internet Access |
|
Holdings |
Library |
Call Number |
Additional Info |
Location |
Last Modified |
Checkout Status |
EJBD |
EPA 600-1-76-001 |
|
Headquarters Library/Washington,DC |
01/17/2014 |
EKBD |
EPA-600/1-76-001 |
|
Research Triangle Park Library/RTP, NC |
06/06/2003 |
ELBD ARCHIVE |
EPA 600-1-76-001 |
Received from HQ |
AWBERC Library/Cincinnati,OH |
10/04/2023 |
ELBD |
EPA 600-1-76-001 |
|
AWBERC Library/Cincinnati,OH |
11/15/2016 |
NTIS |
PB-252 309 |
Some EPA libraries have a fiche copy filed under the call number shown. |
|
07/26/2022 |
|
Collation |
x, 84 pages : graphs ; 28 cm. |
Abstract |
Cardiopulmonary and metabolic responses of 20 adult males (age 19-29) before, during and after a 2-hour exposure to either filtered air or 0.50 ppm ozone under four ambient conditions (25C, 45% rh; 31C, 85% rh; 35C, 40% rh; 40C, 50% rh) were determined. Exercise at 40% of the individual's V02 max was performed from 60-90 min of exposure. There were no cardiovascular changes due to ozone exposure but heart rate increased and stroke volume decreased with increasing heat stress. Rectal, mean body, and mean skin temperature also increased. There was a decrease in vital capacity and total lung capacity due primarily to a reduction of inspiratory capacity following ozone exposure. The combination of heat stress and ozone exposure resulted in significantly greater impairment of pulmonary function. The trachial-bronchial irritation caused by ozone reduces the vital capacity and maximum expiratory flow and this effect is more pronounced when the ozone exposure occurs in a hot environment. |
Notes |
Contract no. EPA 68-02-1723. Includes bibliographical references (pages 47-53). |