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Items Found: 36
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Your Search: (AUTHOR=O Callaghan J. P.)
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1-Methyl-4-Phenyl-1,2,3,6-Tetrahydropyridine (MPTP)-Induced Astrogliosis Does Not Require Activation of Ornithine Decarboxylase.
1-Methyl-4-Phenyl-1,2,3,6-Tetrahydropyridine (MPTP)-Induced Damage of Striatal Dopaminergic Fibers Attenuates Subsequent Astrocyte Response to MPTP.
Acute Exposure of the Neonatal Rat to Tributyltin Results in Decreases in Biochemical Indicators of Synaptogenesis and Myelinogenesis.
Acute Exposure of the Neonatal Rat to Triethyltin Results in Persistent Changes in Neurotypic and Gliotypic Proteins (Journal Version).
Administration of 3,3'-Iminodipropionitrile to the Rat Results in Region-Dependent Damage to the Central Nervous System at Levels Above the Brain Stem.
Assessment of Chemically-Induced Alterations in Brain Development Using Assays of Neuron- and Glia-Localized Proteins.
Assessment of neurotoxicity using assays of neuronglia-localized proteins : chronology and critique /
Assessment of Neurotoxicity: Use of Glial Fibrillary Acidic Protein as a Biomarker.
Biochemical, Functional and Morphological Indicators of Neurotoxicity: Effects of Acute Administration of Trimethyltin to the Developing Rat.
Ca(2+)/Calmodulin-Dependent Protein Phosphorylation Is Not Altered by Amygdaloid Kindling.
Cerebellar Hypoplasia in the Gunn Rat is Associated with Quantitative Changes in Neurotypic and Gliotypic Proteins.
Changes in In vitro Brain and Spinal Cord Protein Phosphorylation After a Single Oral Administration of Tri-o-Cresyl Phosphate to Hens.
Characterization of the Origins of Astrocyte Response to Injury Using the Dopaminergic Neurotoxicant, 1-Methyl-4-Phenyl-1,2,3,6-Tetrahydrophyridine.
Concentration of Glial Fibrillary Acidic Protein Increases with Age in the Mouse and Rat Brain.
Developmental Neurotoxicity: Evaluation of Testing Procedures with Methylazoxymethanol and Methylmercury.
Diethyldithiocarbamate Increases Distribution of Cadmium to Brain but Prevents Cadmium-Induced Neurotoxicity.
Diethyldithiocarbamate Potentiates the Neurotoxicity of In vivo 1-Methyl-4-Phenyl-1,2,3,6-Tetrahydropyridine and of In vitro 1-Methyl-4-Phenylpyridinium.
Effects of 3,4-Methylenedioxymethamphetamine on Autonomic Thermoreregulatory Responses of the Rat.
Enhanced Expression of Glial Fibrillary Acidic Protein and the Cupric Silver Degeneration Reaction Can Be Used as Sensitive and Early Indicators of Neurotoxicity.
Ganglioside Treatment Partially Counteracts Neurotoxic Effects of Trimethyltin but May Itself Cause Neurotoxicity in Rats: Experimental Results and a Critical Review.
Glucocorticoids Regulate the Concentration of Glial Fibrillary Acidic Protein Throughout the Brain.
Glucocorticoids Regulate the Synthesis of Glial Fibrillary Acidic Protein in Intact and Adrenalectomized Rats but Do Not Affect Its Expression Following Brain Injury.
Increase in Glial Fibrillary Acidic Protein Follows Brain Hyperthermia in Rats.
Measurement of Tyrosine Hydroxylase Apoenzyme Protein by Enzyme-Linked Immunosorbent Assay (ELISA): Effects of 1-Methyl-4-Phenyl-1,2,3,6-Tetrahydropyridine (MPTP) on Striatal Tyrosine Hydroxylase Activity and Content.
Nervous-System Specific Proteins as Biochemical Indicators of Neurotoxicity.
Neuron-Specific Phosphoproteins as Biochemical Indicators of Neurotoxicity: Effects of Acute Administration of Trimethyltin to the Adult Rat.
Neurotoxicant 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) Alters Immune Function When Given in Combination with Diethyldithiocarbamate (DDC).
Neurotypic and Gliotypic Proteins as Biochemical Indicators of Neurotoxicity.
Olfactory Toxicity of Beta,Beta'-Iminodipropionitrile in the Rat.
Pargyline and gamma-Butyrolactone Enhance Tyrosine Hydroxylase Immunostaining of Nigrostriatal Axons.
Pharmacologic and Immunologic Approaches to the Problems of Posttraumatic Glial Proliferation Following CNS (Central Nervous System) Damage.
Prolonged Survival of Female AKR Mice Fed Diets Supplemented with Methionine and Choline.
Quantitative Changes in the Synaptic Vesicle Proteins Synapsin I and p38 and the Astrocyte-Specific Protein Glial Fibrillary Acidic Protein are Associated with Chemical-Induced Injury to the Rat Central Nervous System (Journal Version).
Transient Cortical Astrogliosis Induced by Alcohol Exposure during the Neonatal Brain Growth Spurt in Rats.
Use of Glial Fibrillary Acidic Protein in First-Tier Assessments of Neurotoxicity.
Variations in the Neurotoxic Potency of Trimethyltin.
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