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Main Title Fish Physiology, Toxicology, and Water Quality. Proceedings of the Eighth International Symposium, Chongqing, China, October 12-14, 2004.
Author Randall, D. ; Mandy, D. Y. M. ;
CORP Author Environmental Protection Agency, Athens, GA. Ecosystems Research Div.;Environmental Protection Agency, Washington, DC. Office of Research and Development.
Publisher Jun 2006
Year Published 2006
Report Number EPA/600/R-06/062;
Stock Number PB2006-114095
Additional Subjects Meeting ; Fish physiology ; Toxicology ; Water quality management ; Fishes ; Animals ; Water pollution effects(Animals) ; Water quality ; Anoxia ; Hypoxia ; Adenosime ; Ecology ; Pollution ; Climate change ; Eutrophication ; Environmental protection ; Research ; Symposia ; Ion chemistry ; Redox chemistry ; Sediment biota morphology
Internet Access
Description Access URL
Library Call Number Additional Info Location Last
NTIS  PB2006-114095 Most EPA libraries have a fiche copy filed under the call number shown. Check with individual libraries about paper copy. NTIS 05/28/2007
Collation 160p
Abstract Twenty-one participants from Europe, North America and China convened in Chongqing, China, October 12-14, 2005, for the Eighth International Symposium in Fish Physiology, Toxicology and Water Quality. The subject of the meeting was Hypoxia in vertebrates: Comparisons of terrestrial and aquatic vertebrates. These Proceedings include 13 papers presented over the three day period and discuss the responses of fish, reptiles and mammals to hypoxia. These papers report on the responses of animals to hypoxia at the behavioural, physiological and molecular levels. Clearly hypoxia has wide ranging effects, the responses are complex and there are many similarities in the responses of all vertebrates to hypoxia. Organisms respond to hypoxia by reducing energy expenditure, in particular inhibiting reproduction, feeding and exercise. That is animals only expend energy that is absolutely necessary for survival. There is extensive reorganization of cellular machinery directed by hypoxia inducible factors (HIFs), proteins that increase during hypoxia and have a marked effect on the expression of many genes. As a result, anaerobic metabolism is up regulated and many aspects of aerobic metabolism are down regulated. Many cells enter cell cycle arrest. Thus organisms stop reproducing and growing during hypoxia and if the effects of hypoxia are severe, development of eggs and larvae is compromised.
Supplementary Notes See also PB2005-110800. Sponsored by Environmental Protection Agency, Washington, DC. Office of Research and Development.
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Category Codes 68D; 57Y; 57H; 98F
NTIS Prices PC A09
Document Type NT
Cataloging Source NTIS/MT
Control Number 029000800
Origin NTIS
Type CAT