Record Display for the EPA National Library Catalog

RECORD NUMBER: 314 OF 2070

OLS Field Name OLS Field Data
Main Title Chronic Toxicity of Ammonia to Rainbow Trout.
Author Thurston, R. V. ; Russo, R. C. ; Meyn, E. L. ; Luedtke, R. J. ; Smith, C. E. ;
CORP Author Montana State Univ., Bozeman. Fisheries Bioassay Lab.;Environmental Research Lab.-Duluth, MN.
Publisher c1984
Year Published 1984
Report Number EPA-R-800861 ;EPA-R-803950; EPA/600/J-84/425;
Stock Number PB90-106196
Additional Subjects Toxicity ; Ammonia ; Trout ; Blood chemical analysis ; Water analysis ; Tables(Data) ; Mortality ; Pathology ; Reproduction(Biology) ; Liver ; Reprints ; Water pollution effects(Animals) ; Solmo gairdneri ; Gills ; Dose response relationships
Holdings
Library Call Number Additional Info Location Last
Modified
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Status
NTIS  PB90-106196 Most EPA libraries have a fiche copy filed under the call number shown. Check with individual libraries about paper copy. NTIS 03/10/1990
Collation 13p
Abstract
The chronic effects of ammonia to rainbow trout Salmo gairdneri were studied in a laboratory test conducted over a 5-year period. Fish were rested at five concentrations over the range 0.01-0.07 mg/liter un-ionized ammonia: the mean pH of the test water was 7.7, and the mean temperature was 9.3 C. Parental fish were exposed for 11 months, the first filial generation (F1) for 4 years, and the second filial generation (F2) for 5 months. The parental fish spawned of their own volition at all ammonia concentrations tested; baskets containing crushed rock served as the spawning substrate. The F1 fish did not spawn voluntarily at either 3 or 4 years of age, although manual spawning of 4-year-old F1 fish produced viable eggs. There was no significant correlation between ammonia concentration and numbers of egg lots spawned, total numbers of eggs produced, numbers of viable eggs, growth of progeny, or mortality of parents or progeny in any of the generations tested. Blood ammonia concentrations were measured in F1 fish, and proved to be positively correlated with ammonia concentrations in the rest water. Histopathological lesions were common in parental and F1 fish at un-ionized ammonia concentrations of 0.04 mg/liter and higher; in F2 fish, which incurred a severe protozoan infection (Costa sp.), lesions were common at 0.02 mg/liter and higher.