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RECORD NUMBER: 47 OF 67

OLS Field Name OLS Field Data
Main Title Residential indoor exposures of children to pesticides following lawn applications /
Author Lewis, Robert G. ; Nishhioka, M. G.
Other Authors
Author Title of a Work
Nishhioka, Marcia G.
CORP Author Environmental Protection Agency, Research Triangle Park, NC. National Exposure Research Lab. ;Battelle Memorial Inst., Columbus, OH.
Publisher U.S. Environmental Protection Agency, National Exposure Research Laboratory,
Year Published 1999
Report Number EPA/600/A-99/069
Stock Number PB99-175432
OCLC Number 47061208
Subjects Pediatric toxicology. ; Pesticides--Toxicology. ; Children--Health and hygiene. ; Health risk assessment. ; Pesticides--Health aspects. ; Pesticides--Environmental aspects.
Additional Subjects Pesticides ; Children ; Lawns ; Exposures ; Residential buildings ; Indoor air pollution ; Sampling ; Residues ; Floor dust
Holdings
Library Call Number Additional Info Location Last
Modified
Checkout
Status
NTIS  PB99-175432 Most EPA libraries have a fiche copy filed under the call number shown. Check with individual libraries about paper copy. NTIS 01/01/1988
Collation 16 pages ; 28 cm
Abstract
Methods have been developed to estimate children's residential exposures to pesticide residues and applied in a small field study of indoor exposures resulting from the intrusion from the intrusion of lawn-applied herbicide into the home. Sampling methods included size-selective indoor air sampling; wipe sampling of floors, sills, tables; the polyurethane foam (PUF) roller for dislodgeable carpet surface residues; and the HVS3 vacuum sampler for floor dust. Personal exposure samples included hand wipes and morning void urine samples. Pesticide spray drift was found to result in only a relatively minor increase in indoor pesticide levels. post-application air intrusion from closed house ventilation and the opening and closing of doors and windows increased indoor background levels 6-fold, while track-in by high activity children and pets, and wearing shoes indoors, increased indoor levels by 37-fold. Indoor 2,4-D levels were found to increase continually over a one-week period, with the increase in indoor air levels corresponding to the increased floor dust levels, suggesting resuspension of house dust by human activity. Similar estimates of non-dietary exposure are obtained from models based on 100 mg ingestion and surface contact simulated by the PUF roller.
Notes
EPA/600/A-99/069. Cover title. Microfiche.