||Human population exposures to Mirex and Kepone /
Suta, Benjamin E.
||Stanford Research Inst., Menlo Park, Calif. Center for Resource and Environmental Systems Studies.;Environmental Protection Agency, Washington, D.C. Office of Research and Development.
|| U.S. Environmental Protection Agency, Office of Research and Development,
||EPA/600/1-78/045; CRESS-26; EPA-68-01-4314
Chlordecone--Toxicology--United States. ;
Mirex--Toxicology--United States. ;
Chlordecone--Environmental aspects--United States. ;
Environmental Pollution. ;
Hydrocarbons, Chlorinated. ;
Chlordecone--Toxicology--United States ;
Mirex--Toxicology--United States ;
Chlordecone--Environmental aspects--United States ;
Hazardous materials ;
Chlorine alaphatic compounds ;
Chlorine organic compounds ;
Air pollution ;
Water pollution ;
Potable water ;
Food chains ;
Food consumption ;
United States ;
Toxic substances ;
Environmental exposure pathway ;
Environmental persistence ;
Pesticide residues ;
||Region 3 Library/Philadelphia, PA
||OCSPP Chemical Library/Washington,DC
||Most EPA libraries have a fiche copy filed under the call number shown. Check with individual libraries about paper copy.
||ix, 139 p. ; 28 cm.
Human exposures to mirex and kepone are assessed in this study. Three major paths of exposure are examined: ingestion through the food chain, inhalation of atmospheric mirex and kepone, and exposure through drinking water. Exposure through the food chain appears to be the most pressing current problem. Various species of commercially caught fish in certain areas of the country have been found to contain slight amounts of the compounds. It is difficult to obtain estimates of the human exposure from sport fish, but evidence seems to suggest that sportsmen and their families may be exposed through fish. Also, game captured in the southeastern U.S. has been found to contain the compounds. Kepone has been found in mothers' milk in some areas of the country. Atmospheric exposures are not considered terribly great now, since the compounds are no longer produced and the major atmospheric exposures are believed to be occupational exposure at the producing factory and exposure to workers' families from clothing, etc. Human exposure to mirex and kepone from drinking water supplies does not appear to be a problem, since both compounds are very insoluble in water. Exposure through tobacco was also considered.
Bibliography: p. 133-139.