Record Display for the EPA National Library Catalog

RECORD NUMBER: 5 OF 11

OLS Field Name OLS Field Data
Main Title Effects of Sodium Pentachlorophenate on Survival and Energy Metabolism of Embryonic and Larval Steelhead Trout.
Author Chapman, Gary A. ; Shumway, Dean L. ;
CORP Author Corvallis Environmental Research Lab., OR. ;Federal Power Commission, Washington, DC. Bureau of Power.
Year Published 1978
Report Number EPA/600/J-78/051;
Stock Number PB-287 600
Additional Subjects Fishes ; Trout ; Toxicology ; Embryos ; Experimental data ; Mortality ; Concentration(Composition) ; Exposure ; Lethal dosage ; Metabolism ; Physiological effects ; Fresh water fishes ; Chlorine organic compounds ; Bioassay ; Tables(Data) ; Oxygen consumption ; Reprints ; Steelhead trout ; Salmo gairdnerii ; Sodium pentachlorophenate ; Toxic substances ; Water pollution effects(Animals)
Holdings
Library Call Number Additional Info Location Last
Modified
Checkout
Status
NTIS  PB-287 600 Most EPA libraries have a fiche copy filed under the call number shown. Check with individual libraries about paper copy. NTIS 06/23/1988
Collation 17p
Abstract
A study was conducted to determine the effects of technical grade sodium pentachlorophenate (Na-PCP) on the early developmental stages of the steelhead trout (Salmo gairdneri). In an experiment where embryos were exposed to Na-PCP from fertilization to hatching, 100% mortality occurred within one week after fertilization at concentrations down to 300 ppb (g/l); within 24 hours posthatch, 100% mortality occurred down to 50 ppb of Na-PCP. Alevin dry weight at hatch was decreased by exposure to Na-PCP and hatching was delayed. In 5-day tests, alevins usually died within 24 hours at concentrations down to 200 ppb, but little mortality occurred at lower concentrations. Continuous exposure to Na-PCP from fertilization to complete yolk absorption produced 100% mortality at 40 ppb Na-PCP but little mortality at 20 or 10 ppb. However, in water containing 5 mg O2/l, 20 ppb Na-PCP was 100% lethal and at 3 mg O2/l, 10 ppb was 100% lethal. Little mortality occurred at these oxygen levels in the absence of Na-PCP. Oxygen consumption rates of alevins in 40 ppb Na-PCP was higher than those of control alevins. Exposure to Na-PCP reduced yolk utilization efficiency and growth. The bioenergetic data obtained in the study are consistent with the concept that PCP disrupts energy metabolism.