Record Display for the EPA National Library Catalog


OLS Field Name OLS Field Data
Main Title Effects of Sodium Pentachlorophenate on Survival and Energy Metabolism of Embryonic and Larval Steelhead Trout.
Author Chapman, Gary A. ; Shumway, Dean L. ;
CORP Author Corvallis Environmental Research Lab., OR. ;Federal Power Commission, Washington, DC. Bureau of Power.
Year Published 1978
Report Number EPA/600/J-78/051;
Stock Number PB-287 600
Additional Subjects Fishes ; Trout ; Toxicology ; Embryos ; Experimental data ; Mortality ; Concentration(Composition) ; Exposure ; Lethal dosage ; Metabolism ; Physiological effects ; Fresh water fishes ; Chlorine organic compounds ; Bioassay ; Tables(Data) ; Oxygen consumption ; Reprints ; Steelhead trout ; Salmo gairdnerii ; Sodium pentachlorophenate ; Toxic substances ; Water pollution effects(Animals)
Library Call Number Additional Info Location Last
NTIS  PB-287 600 Most EPA libraries have a fiche copy filed under the call number shown. Check with individual libraries about paper copy. 06/23/1988
Collation 17p
A study was conducted to determine the effects of technical grade sodium pentachlorophenate (Na-PCP) on the early developmental stages of the steelhead trout (Salmo gairdneri). In an experiment where embryos were exposed to Na-PCP from fertilization to hatching, 100% mortality occurred within one week after fertilization at concentrations down to 300 ppb (g/l); within 24 hours posthatch, 100% mortality occurred down to 50 ppb of Na-PCP. Alevin dry weight at hatch was decreased by exposure to Na-PCP and hatching was delayed. In 5-day tests, alevins usually died within 24 hours at concentrations down to 200 ppb, but little mortality occurred at lower concentrations. Continuous exposure to Na-PCP from fertilization to complete yolk absorption produced 100% mortality at 40 ppb Na-PCP but little mortality at 20 or 10 ppb. However, in water containing 5 mg O2/l, 20 ppb Na-PCP was 100% lethal and at 3 mg O2/l, 10 ppb was 100% lethal. Little mortality occurred at these oxygen levels in the absence of Na-PCP. Oxygen consumption rates of alevins in 40 ppb Na-PCP was higher than those of control alevins. Exposure to Na-PCP reduced yolk utilization efficiency and growth. The bioenergetic data obtained in the study are consistent with the concept that PCP disrupts energy metabolism.