| Main Title |
Subacute Ethanol Consumption Reverses p-Xylene-Induced Decreases in Axonal Transport. |
| Author |
Padilla, S. ;
Lyerly, D. L. ;
Pope, C. N. ;
|
| CORP Author |
Environmental Protection Agency, Research Triangle Park, NC. Neurotoxicology Div. ;Burroughs Wellcome Co., Research Triangle Park, NC. ;Northeast Louisiana Univ., Monroe. Toxicology Program. |
| Publisher |
c1992 |
| Year Published |
1992 |
| Report Number |
EPA/600/J-92/425; |
| Stock Number |
PB93-141240 |
| Additional Subjects |
Toxicology ;
Xylenes ;
Ethyl alcohol ;
Axons ;
Nervous system ;
Rats ;
Neurons ;
Protein synthesis ;
Environmental exposure ;
Reprints ;
Axonal transport
|
| Holdings |
| Library |
Call Number |
Additional Info |
Location |
Last
Modified |
Checkout
Status |
| NTIS |
PB93-141240 |
Some EPA libraries have a fiche copy filed under the call number shown. |
|
07/26/2022 |
|
| Collation |
11p |
| Abstract |
Organic solvants, as a class, have been implicated as neurotoxic agents in humans and laboratory animals. The study was designed to assess the interaction between subacute ingestion of moderate levels of ethanol and the p-xylene-induced decreases in protein and glycoprotein synthesis and axonal transport in the rat optic system. The results indicated that animals maintained on 10% ethanol as a drinking liquid show less p-xylene-induced neurotoxicity than animals receiving no ethanol supplement. |
