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Main Title Handbook of Neurochemistry and Molecular Neurobiology Schizophrenia / [electronic resource] :
Author Lajtha, Abel.
Other Authors
Author Title of a Work
Javitt, Daniel.
Kantrowitz, Joshua.
Publisher Springer US : Imprint: Springer,
Year Published 2009
Call Number RC321-580
ISBN 9780387304106
Subjects Medicine ; Neurosciences ; Psychology, clinical
Internet Access
Description Access URL
Collation XII, 549 p. eReference. online resource.
Due to license restrictions, this resource is available to EPA employees and authorized contractors only
Contents Notes
Hippocampus -- Redox dysregulation in schizophrenia: Genetic susceptibility and pathophysiological mechanisms -- An overview and current perspective on family studies of schizophrenia -- Synaptic vesicle associated proteins and schizophrenia -- Cortical dopamine in schizophrenia -- G72/g30 in neuropsychiatric disorders -- Cholinergic mechanisms in schizophrenia -- The neurobiology of negative symptoms and the deficit syndrome -- Alterations of neurotransmitter receptors in schizophrenia: Evidence from postmortem studies -- Auditory cortex anatomy and asymmetry in schizophrenia -- Event related potentials (ERPS) in the study of schizophrenia: How pre-clinical ERP studies have contributed to our understanding of schizophrenia -- Neuregulin 1 and schizophrenia -- The role of prefrontal abnormalities in schizophrenia -- Early-stage visual processing deficits in schizophrenia and implications for higher level function -- Magnetic resonance spectroscopy -- Dysbindin-1 and its protein family -- Glutamate and schizophrenia: Phencyclidine, n-methyl-d-aspartate (NMDA) receptors: Etiology and treatment. This volume of the Handbook of Neurochemistry and Molecular Neurobiology focuses on neurochemical aspects of schizophrenia. Chapters cover the full range of schizophrenia symptoms and anatomical pathologies from neurochemical and molecular biology perspectives. Topics include changes in neurotransmitter systems, alteration in receptors, neurotransmitter release, genetic factors, protein alterations, and redox dysregulation.