||Nitrogen oxides /
||National Research Council, Washington, D.C. Committee on Medical and Biologic Effects of Environmental Pollutants.;Health Effects Research Lab., Research Triangle Park, N.C.
|| National Academy of Sciences,
||EPA 600/1-77-013; EPA-68-02-1226; PB264872
Nitrogen Oxides--toxicity. ;
Nitrogen Oxides--adverse effects. ;
Nitrogen oxides ;
Air pollution ;
Physical properties ;
Chemical properties ;
Combustion products ;
Industrial wastes ;
Fossil fuels ;
Public healths ;
Photochemical reactions ;
Mathematical models ;
Air pollution control ;
Chemical reactions ;
Light transmission ;
Atmospheric composition ;
Urban areas ;
Reaction kinetics ;
Gas analysis ;
Chemical analysis ;
Exhaust emissions ;
Electric power plants ;
Chemical industry ;
Air pollution effects(Humans) ;
Air pollution effects(Plants) ;
Air pollution effects(Materials) ;
Atmospheric chemistry ;
Air pollution detection ;
||Research Triangle Park Library/RTP, NC
||Region 9 Library/San Francisco,CA
||Some EPA libraries have a fiche copy filed under the call number shown.
||viii,  p. in various pagings : ill. ; 28 cm.
This report is a review of current knowledge of the environmental health basis for control of manmade sources of nitrogen oxide emissions. The literature review covered the period through 1974. The principal subject areas considered in the report include: sources and control of atmospheric nitrogen oxides; analytical methodology; concentrations and chemical reactions in the atmosphere; and the effects of nitrogen oxides on human health, materials, vegetation, light transmission, and natural ecosystems. Emphasis is primarily on nitric oxide (NO) and nitrogen dioxide (NO2), designated by the composite formula NOx for nitrogen oxides. The major manmade source is the combustion of fossil fuel. Highest atmospheric concentrations are found in heavily populated, industrialized urban areas. Both acute and chronic health effects resulting from short-term and long-term exposures, are discussed in the report. Effects range from slight increases in airway resistance to death depending upon exposure concentrations.
Includes bibliographical references.