| Main Title |
Growth and Dissipation of Pesticide Oxons. |
| Author |
Stamper, James H. ;
Nigg, Herbert N. ;
Winterlin, Wray ;
|
| CORP Author |
Agricultural Research and Education Center, Lake Alfred, FL.;Health Effects Research Lab., Research Triangle Park, NC. |
| Year Published |
1981 |
| Report Number |
EPA-R-806474; EPA-600/J-82-210; |
| Stock Number |
PB82-251265 |
| Additional Subjects |
Phosphorous organic compounds ;
Industrial hygiene ;
Predictions ;
Reprints ;
Pesticide residues ;
Oxons
|
| Holdings |
| Library |
Call Number |
Additional Info |
Location |
Last
Modified |
Checkout
Status |
| NTIS |
PB82-251265 |
Some EPA libraries have a fiche copy filed under the call number shown. |
|
07/26/2022 |
|
| Collation |
8p |
| Abstract |
Progress toward a solution to the worker reentry problem requires a method for predicting the conversion of applied organophosphate pesticides to their oxon analogs, together with subsequent oxon persistence. That oxons are more toxic than their parent compound is well known. For examle, paraoxon is ten times more toxic intravenously and fifty-five times more toxic dermally than the same amount of parathion (NABB et.al. 1966). In some regions of the United States, it is not unusual for the oxon level to exceed that of the parent at some point in the growth-decay process of the oxon on the plant or soil surface. |
